Pinna AVM embolisation
A young man presented with pulsatile swelling of the left pinna, with reddish brown skin discoloration.
He was diagnosed to have arteriovenous malformation and was taken up for DSA and embolisation under local anesthesia.
DSA showed a high flow AVM with a large fistula. This was then catheterized with a Progreat 2.7 Fr microcatheter, considering the large size of the feeder and the macro-fistula. Embolisation was done with 20% nBCA-lipiodol mixture. Complete exclusion of the AVM was achieved.
There was subsequently blackening of the skin of the earlobe, which necrosed and fell off, it was conservatively managed, with normal smooth skin formation over the ulcerated area and near comparable appearance with the opposite pinna. However there was some redness present in the mid-pinna, which was not explained, and was left behind. This patient is now in follow up since three years.
He was diagnosed to have arteriovenous malformation and was taken up for DSA and embolisation under local anesthesia.
DSA showed a high flow AVM with a large fistula. This was then catheterized with a Progreat 2.7 Fr microcatheter, considering the large size of the feeder and the macro-fistula. Embolisation was done with 20% nBCA-lipiodol mixture. Complete exclusion of the AVM was achieved.
There was subsequently blackening of the skin of the earlobe, which necrosed and fell off, it was conservatively managed, with normal smooth skin formation over the ulcerated area and near comparable appearance with the opposite pinna. However there was some redness present in the mid-pinna, which was not explained, and was left behind. This patient is now in follow up since three years.
Microcatheter injection, showing the AVF
Microcatheter injection, late phase, showing the nidus
Post embolisation, control angiography, showing complete exclusion of the AVM
Glue cast, fluorospot image
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Post Biopsy pseudoaneurysm embolisation
A middle aged male with Lymphoma had undergone CT guided retroperitoneal lymph node biopsy.
Subsequently, after 4 hours he started complaining of severe radiating pain.
A CT was done which showed a pseudoaneurysm from the lumbar artery wiht a large psoas hematoma.
Immediately the patient was taken up for DSA which showed the pseudoaneurysm from the right first lumbar artery branch.
This was embolised using nBCA-lipiodol mixture and microcoil.
Subsequently, the hematoma resolved.
Subsequently, after 4 hours he started complaining of severe radiating pain.
A CT was done which showed a pseudoaneurysm from the lumbar artery wiht a large psoas hematoma.
Immediately the patient was taken up for DSA which showed the pseudoaneurysm from the right first lumbar artery branch.
This was embolised using nBCA-lipiodol mixture and microcoil.
Subsequently, the hematoma resolved.
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How fast can flow diverters act ?
Flow diverters are used for many purposes:
1. Giant aneurysms
2. Dissecting aneurysms
3. Blister aneuryms
4. Fusiform aneuryms
5. Wide neck aneurysms
While many cases have contrast stasis immediately after flow diverter placement, actual aneurysm 'closure' and arterial wall remodeling and endothelialisation takes place after a variable time-frame.
We came across a case of subarachnoid hemorrhage, wherein DSA showed a irregular mild fusiform dilatation of the supraclinoid ICA with multiple blister like outpouchings.
Single PED Flex was placed, with no immediate change in appearance.
Patient developed vasospasm features, and was taken up for intra-arterial spasmolysis, during which the DSA showed smoothing of the arterial contour and non-visualisation of the blisters.
It can be said with guarantee that endothelialisation has not taken place, and the smoothening is due to the flow diversion effect. Probably the vasospasm also has added to the appearance.
DIAGNOSTIC DSA
FLOW DIVERTER
NEXT MORNING CHECK ANGIOGRAM
1. Giant aneurysms
2. Dissecting aneurysms
3. Blister aneuryms
4. Fusiform aneuryms
5. Wide neck aneurysms
While many cases have contrast stasis immediately after flow diverter placement, actual aneurysm 'closure' and arterial wall remodeling and endothelialisation takes place after a variable time-frame.
We came across a case of subarachnoid hemorrhage, wherein DSA showed a irregular mild fusiform dilatation of the supraclinoid ICA with multiple blister like outpouchings.
Single PED Flex was placed, with no immediate change in appearance.
Patient developed vasospasm features, and was taken up for intra-arterial spasmolysis, during which the DSA showed smoothing of the arterial contour and non-visualisation of the blisters.
It can be said with guarantee that endothelialisation has not taken place, and the smoothening is due to the flow diversion effect. Probably the vasospasm also has added to the appearance.
DIAGNOSTIC DSA
FLOW DIVERTER
NEXT MORNING CHECK ANGIOGRAM
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